Please use this identifier to cite or link to this item: https://hdl.handle.net/10137/6930
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dc.contributor.authorSolomon, M Jen
dc.contributor.authorStening, M Ren
dc.contributor.authorHargrave, J. C. (John C.)en
dc.date.accessioned2018-05-24T23:55:40Zen
dc.date.available2018-05-24T23:55:40Zen
dc.date.issued1990-07en
dc.identifier.citationThe Australian and New Zealand journal of surgery 1990-07; 60(7): 545-8en
dc.identifier.issn0004-8682en
dc.identifier.urihttps://hdl.handle.net/10137/6930en
dc.description.abstractExtrahepatic portal hypertension was induced in the rabbit by a one-stage complete ligation of the portal vein. End renal vein to side splenic vein shunts (renosplenic) were performed with haemorrhagic necrosis of the left kidney occurring after ligation of the left renal vein lateral to the adrenolumbar tributary. Thus the ureteric, lumbar, and pericapsular collaterals cannot adequately drain the left kidney. Ligation of the left renal vein on the medial side of the adrenolumbar tributary maintained a patent left renal vein in all cases with 60% of left kidney biopsies showing no histological evidence of changes to glomeruli or tubules, and the remainder showing early acute tubular necrosis.en
dc.language.isoengen
dc.titleThe fate of the left kidney after end renosplenic shunt in experimentally induced extrahepatic portal hypertension.en
dc.typeJournal Articleen
dc.identifier.journaltitleThe Australian and New Zealand journal of surgeryen
dc.identifier.pubmedidhttps://www.ezpdhcs.nt.gov.au/login?url=https://www.ncbi.nlm.nih.gov/pubmed//2357180en
dc.subject.meshAnimalsen
dc.subject.meshHypertension, Portalen
dc.subject.meshKidneyen
dc.subject.meshKidney Tubular Necrosis, Acuteen
dc.subject.meshLigationen
dc.subject.meshPortal Veinen
dc.subject.meshRabbitsen
dc.subject.meshRenal Circulationen
dc.subject.meshRenal Veinsen
dc.subject.meshSplenic Veinen
dc.identifier.affiliationDepartment of Surgery, Royal Darwin Hospital, Casuarina, Northern Territory, Australia..en
dc.identifier.pubmedurihttps://www.ezpdhcs.nt.gov.au/login?url=https://www.ncbi.nlm.nih.gov/pubmed//2357180en
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